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AML with t(8; 21)

View larger version (114K): [in this window] [in a new window]   Figure 1. Blast circulating in the peripheral blood.

View larger version (99K): [in this window] [in a new window]   Figure 2. Auer rods (arrow) are common in this disorder and tend to occur as single thin structures within the cytoplasm of the blasts.

View larger version (94K): [in this window] [in a new window]   Figure 3. Cytoplasm of some of the blasts may be intensely basophilic. Nuclei may be indented.

View larger version (89K): [in this window] [in a new window]   Figure 4. Arrow marks "thumbprinting" which is characteristic of myeloid blasts.

View larger version (78K): [in this window] [in a new window]   Figure 5. Few faint granules may be present in the cytoplasm of the blasts.

View larger version (165K): [in this window] [in a new window]   Figure 6. Blasts are increased in the bone marrow and,although many of the cells appear to have a centrosome, maturation is arrested.

View larger version (123K): [in this window] [in a new window]   Figure 7. Some blasts have prominent nucleoli.

View larger version (121K): [in this window] [in a new window]   Figure 8. Centrosomes are evidence of myeloid differentiation.

View larger version (120K): [in this window] [in a new window]   Figure 9. Some of the nuclei are folded and somewhat irregular.

View larger version (78K): [in this window] [in a new window]   Figure 10. Diagram of the t(8;21)(q22;q22) rearrangement which characterizes this disorder and results in the molecular fusion of part of the AML1 gene at 8q22 with part of the ETO gene from 21q22.

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